Poster Presentation & Flash Talk Presentation 28th Lorne Cancer Conference 2016

Harnessing functional genomics to identify novel microRNA-based combination therapies in cancer (#221)

Iva Nikolic 1 2 , Ben Elsworth 1 3 , Eoin Dodson 1 , Kate Gould 1 , Vinod Ganju 4 , Jennifer MacDiarmid 5 , Kaylene Simpson 2 , Alexander Swarbrick 1
  1. The Garvan Institute, Darlinghurst, NSW, Australia
  2. Victorian Centre for Functional Genomics and Proteomics, Peter MacCallum Cancer Centre, Melbourne, Victoria, Australia
  3. Integrative Cancer Epidemiology Programme (ICEP), MRC Integrative Epidemiology Unit, Bristol, UK
  4. Peninsula Oncology Centre, Melbourne, VIC, Australia
  5. EnGeneIC Pty Ltd, Sydney, NSW, Australia
Despite considerable success in developing novel targeted therapies drug resistance is still the largest obstacle for more effective management of cancer. For many types of cancer there is also paucity of clearly defined molecular targets, and chemotherapy remains the only treatment option available. This calls for rethinking of how we study cancer chemoresistance and for devising alternative treatment strategies, enabling better treatment outcomes. MicroRNAs are small non-coding RNAs able to simultaneously regulate hundreds of targets that operate within complex functional programs important for cancer development and maintenance. MicroRNA-based drugs therefore represent an exciting new therapy approach, and several microRNA mimics and inhibitors have been successfully tested in clinical trials. Here we employ functional genomics to search for the most potent microRNA candidates that either kill cancer cells on their own or synergise with different chemotherapeutic drugs. We performed genome-wide inhibition and overexpression of ~1500 microRNAs in breast cancer, neuroblastoma, and prostate cancer models in vitro, in the presence of common molecularly distinct chemotherapeutic drugs. Cell viability measurements revealed numerous cell-type- and drug-type-specific synthetic lethal interactions between the microRNAs and chemotherapy. Different functional assays, in addition, enabled us to pinpoint the exact cellular processes—such as apoptosis, proliferation, or DNA damage—that underlie these effects. Finally, to search for direct microRNA targets, we measured changes in gene expression in cells expressing candidate microRNA mimics and constructed novel tools for microRNA target prediction and prioritisation. This approach identified several strong candidates, which we are currently pursuing in vivo using an innovative method of targeted drug delivery. We believe that our comprehensive and integrative analysis of microRNA function will be an important resource both for studying the mechanism of drug resistance and for identifying the most effective microRNA-based combination therapies.